Samer Hattar - Recent Papers

Hattar, S., Kumar, M., Park, A., Tong, P., Tung, J., Yau, K.-W., Berson, D.M. (2006) Central Projections of Melanopsin-Expressing Retinal Ganglion Cells in the Mouse.Journal of Comparative Neurology 497:326-349. [PDF]

Sekaran, S., Lupi, D., Jones, S.L., Sheely, C.J., Hattar, S., Yau, K.-W., Lucas, R.J., Foster, R.G., and Hankins, M.W., Melanopsin-Dependent Photoreception Provides Earliest Light Detection in the Mammalian Retina. Curr Biol, Published online May. 26, 2005.

Hattar, S., Lucas, R.J. Mrosovsky, N., Thompson, S., Douglas, R.H., Hankins, M.W., Lem, J., Biel, M., Hofmann, F., Foster, R.G., and Yau, K.-W., (2003). Melanopsin and Rod–Cone Photoreceptive Systems Account for All Major Accessory Visual Functions in Mice. Nature 424, 76-81. Advance Online Publication, 15 June 2003 (doi:10.1038/nature01761). [PDF]

Lucas, R.J.*, Hattar, S.*, Takoa, M., Berson, D.M., Foster, R.G., and Yau K.-W., (2003). Diminished Pupillary Light Reflex at High Irradiances in Melanopsin-Knockout Mice, Science 299, 245–247. * Authors contributed equally to this work. (See related perspective page 213; cover). [PDF]

Hattar, S.*, Liao, H.-W.*, Takoa, M., Berson, D.M., and Yau, K.-W. (2002). Melanopsin-Containing Retinal Ganglion Cells: Architecture, Projections, and Intrinsic Photosensitivity. Science 295, 1065-70. * Authors contributed equally to this work. (See News Focus page 955). [PDF]

Hattar, S., Lyons, L.C., Dryer, L., and Eskin, A. (2002). Circadian Regulation of the Transcription Factor, ApC/EBP in the Eye of Aplysia Californica. J Neurochem 83, 1401-11. [PDF]

Zwartjes, R.E., West, H., Hattar, S., Ren, X., Noel, F., Nuñez-Regueiro, M., MacPhee, K., Homayouni, R., Crow, M.T., Byrne, J.H., and Eskin, A. (1998). Identification of Specific mRNAs Affected by Treatments Producing Long-Term Facilitation in Aplysia. Learning & Memory 4, 478-495. [LINK]

Liu, Q.R., Hattar, S., Endo, S., MacPhee, K., Zhang, H., Cleary, L.J., Byrne, J.H., and Eskin, A. (1997). A Developmental Gene (Tolloid/BMP-1) is Regulated in Aplysia Neurons by Treatments that Induce Long-Term Sensitization. J Neurosci. 17, 755-764. [PDF]